The Hippocampus Release Event and Reset Process Technique – Overview

Summary

The hippocampus is an organ in the brain which, amongst other functions, is responsible for the formation and storage management of our lifetime memories. It is also connected with epilepsy.

The hippocampus release event and reset process described in the first section appears to be a naturally occurring phenomenon of human physiology. The release event is a spontaneous process in the brain which is part of our inherent capability for brain tissue repair and regeneration. It is a specific instance of the natural clearance and reset of an organ in the brain at the conclusion of an extreme and sustained human fear cycle, and can be triggered by the release of the soft tissue head injuries which are preventing the closure of an active fear cycle. The event requires conscious physical participation to prepare for its neural release, so it can only happen while you are awake and alert.

The release triggers a reset of multiple neural pathways in brain tissue which can have profoundly beneficial life-changing consequences. The first phase of the reset process takes about six weeks to complete. Further and more gentle behavioural changes become apparent during the second phase of the reset, which can take up to about six months.

The hippocampus resets by purging itself of newly-redundant memories and their associated emotions at the end of its most recent active human fear cycle, and does do in partnership with the hypothalamus and the amygdala. During the heat-based release process, both the internal photograph and video albums of individual traumatic events managed by the hippocampus and their related emotional content managed by the amygdala appear to be deleted from our memory’s index catalogues. These de-catalogued memories subsequently disintegrate physically and spontaneously in the cortex, where their neural and chemical clusters are stored.

The physiology of the injury release which triggers the hippocampus release event indicates that a human fear cycle can stay active indefinitely following a traumatic event, until the physical injuries preventing its conclusion are resolved.

The summary neuroscience which may underpin this injury release technique is outlined in the second section. This section also describes how survivors of impact trauma may unknowingly be living in a constantly active state of fear, despite the apparent conclusion of their traumatic experiences, and discusses how we may individually be able to take control of an active human fear cycle and turn it off for ourselves.

The Building Blocks of The Release Event and Reset Process (1)

The organs in the brain which could be involved in the release process include:

How Traumatic Memories Are Created In The First Place

The hippocampus ‘dashcam’ is the memory function which records every event which occurs while we are in a heightened state of fear response. It may do this as part of a learning process to inform future self-preservation decisions. The amygdala simultaneously records any significant emotional content of those events, so when we recall these memories after the event we can also remember the accompanying emotional trauma of the incident.

How Does The Technique Work?

Please Note:

You can achieve a hippocampus release event by yourself in private, but it is a specific head trauma release and there is absolutely no loss of face in asking for professional medical support with this, if you would prefer.

Triggering A Release Event

My hippocampus release event started with the spontaneous resolution of two minor facial injuries which I had unknowingly managed to conceal for over sixty years. As part of a lifelong ambition to have another go at singing jazz, I attended a vocal release course. Two of the warm-up exercises, described at the end of this page, released the minor dislocation of my right jaw hinge (temporomandibular joint) and the partial muscular paralysis of my tongue root.

These head injury releases appeared to trigger the hippocampus release event by relieving their indirect distortion of the soft tissue in the brainstem in or around the part of the spinal cord through which a specific neural reflex arc passes.

If there is no apparent jaw or tongue mobility impairment to release, standard KAIR Technique injury releases could be used on any localised soft tissue trauma at the back of the neck. These could substitute for the neural effect of the jaw hinge release by relieving any indirect soft tissue tension in the region of the reflex arc.

This reflex, possibly originating from a defensive survival reflex, could act as the controlling mechanism for the activation of the human fear cycle. The binary (active or not active) reflex ‘trigger’ position could be reset by the transmission of a nerve impulse through the reflex arc to complete its last active cycle. This event could also send a simultaneous nerve signal through the spinal cord to the limbic core of the brain to advise that the most recent vital threat has been vanquished, and the current active fear cycle may now end.

This would be the signal for the autonomic nervous system to stop activating its fear responses, and for the hippocampus’s management system to start preparations for its forthcoming minor or major heat release. A minor fear alert may not result in a noticeable heat release, but the conclusion of an extended and sustained active fear cycle probably will.

What The Heat Release Does

Memories are formed in brain tissue as physical clusters of chemicals, covalently bonded by their electrons. These chemical clusters are linked together by networks of chains of neuron cells, which relay streams of electrons as electrical nerve impulses along their communication pathways. These two components combine to create a physical memory cluster in the brain’s cortex. The process of memory recall is initiated as a nerve impulse crosses a synaptic bridge from the hippocampus to the memory cluster in the cortex.

The hippocampal heat release appears to disable these synaptic bridges, thereby making future recall and replay of each traumatic memory impossible. The detail of the exact way in which the de-cataloguing and physical erasure of traumatic memories takes place remains to be established. The heat release could deform or eliminate the chemical neurotransmitters which relay electrical nerve impulses across the synaptic bridge from the hippocampus to the cortex.

My intuitive suggestion is that the heat release could disrupt the patterns of neurons in the hippocampus (2) which act as library index cards or QR barcodes, linking to the neural storage addresses in the cortex where each long term memory cluster is found. The ‘index card’ would be the physical component of the traumatic memory which vaporises during the heat release, while the memory cluster in the cortex would be the one which physically disintegrates over time. The loss of its ‘index card’ could be enough to destabilise the physical configuration of the newly de-catalogued memory cluster in the cortex, and this could be the event which triggers its physical disintegration.

Interestingly, the de-cataloguing and subsequent deletion of memory process does not appear to affect non-traumatic memory. This suggests that a specific chemical marker which indicates emotional trauma could be attached to the synaptic bridge of each traumatic memory cluster by the amygdala while the memory cluster is being created.

Do I Need A Hippocampus Release and Reset?

All it takes is one physical blow or one moment of abject terror to receive an injury which lasts a lifetime.

If you have ever survived even just one instance of physical and/ or mental abuse, it is possible that you might benefit from this release process. If you have endured a more extended period of abuse, the likelihood of your need increases. If you have ever taken a concussive blow to the back of your head you might particularly need this release process, because the blow’s directional impact could partially dislocate a jaw hinge. There are other head injuries, including facial injuries, which could cause the same indirect muscle tension in the back of the neck which creates the need for the release process.

There may be emotional and behavioural indicators of an inappropriately active fear cycle. We may feel that everything we do in every aspect of our lives is done in a heightened state of fear alert. We are constantly being misinformed by our limbic system that we are facing an existential threat, simply because of the malfunction of the trigger which manages our fear response setting. Our conscious logical brain can override this setting to some extent,  but normal everyday activities such as taking an underground train to go to work, putting petrol in the car, shopping in a supermarket – things we cannot afford to get wrong if we are to survive by getting to work to earn money, and by buying food – transform from mundane to intimidating tasks, about which we may feel consciously apprehensive.

Another cause of these injuries is that they can be inadvertently self-inflicted in a moment of terror, as we perform an intuitive self-protective sequence of physical movements in the face of approaching danger. This sequence of movements is known as the ‘startle’ reflex, or anticipatory bracing (3). We cower, and hunch our shoulders to protect the back of our head and neck. We clench our teeth to prevent dental injury, and wince and grimace to protect our eyes by minimising their physical exposure to threat. These movements also unfortunately cause extreme soft tissue compression in the head, neck and back. This sequence of movements, particularly clenching the teeth or wincing, could quite easily and unnoticeably partially dislocate a jaw hinge and disturb the muscular balance of the tongue, particularly when performed forcefully in a moment of terror. These injuries may not be recognised consciously as they happen because of your speed of response to vital threat. Your conscious brain is prioritising your immediate escape from vital threat, so the pain alerts of these injuries will be subliminally suppressed. The immediate single effect of this injury, though, is simple. The now permanent indirect soft tissue compression jams our fear response trigger setting at ‘active’, and keeps it that way until the injury is released.

The jaw dislocation is minor and almost indetectable, as it does not noticeably inhibit eating or speaking. The problem is that it prevents the jaw from closing completely on both sides, thereby preventing the completion of the full bite action. The injury may be indicated by grinding the teeth while sleeping, by chewing consistently on one side or by biting the side of the mouth or tongue more than infrequently whilst eating.

The tongue root injury is most easily identified if you are regularly accused of speaking too quietly, even though the sound you hear internally seems adequately loud. Further possible indicators could be experiencing momentary and intermittent difficulties with swallowing or choking, or if your tongue’s most convenient resting place usually appears to be the roof of your mouth. The easiest way to identify whether you have these injuries is to try the speech therapy exercises and see whether they make a difference in terms of tongue placement and relaxation and vocal projection.

A further possible indicator of an active fear cycle is invariantly dilated eye pupils. This condition is traditionally diagnosed as a possible indication of a head injury. However, the eye pupils will remain dilated so long as the fear cycle is active, and this can be for decades – long after any possibly causative head injury is assumed to have resolved. Consequently, another possible indicator of this visual injury could be the early onset of dry macular degeneration, since the retinal tissue cells at the back of the eye could have suffered long-term over-exposure to light. Unfortunately, this visual injury is open to misinterpretation, as temporarily invariant eye pupil dilation can also result from the use of chemical stimulants. Furthermore, transitory pupil dilation expresses a positive emotional appreciation of what the owner of the eyes is looking at, whether human or inanimate, but since we usually only glance at someone’s eyes as we speak to them, we may not give ourselves time to notice that the receiving eyes are invariantly and permanently dilated.

The Vocal Release Exercises

The exercises which released my jaw hinge use speech therapy techniques (4). The first exercise involves the repetition of simple monosyllabic sounds using the letter ‘v’, and are specifically designed to loosen the jaw. The sequence of sounds is:

Va Vay  Va Vay  Va Vay  Va Vay       Vee Vay  Va Va  Vee Vay  Voo Vo Vumm

The sounds are pronounced at a reasonable pace as you smile in a slightly surprised way, since this mouth position securely anchors the jaw hinges in place. Further advice is to keep the jaw light and bouncy and the vowels smooth and clear.

A supplementary jaw loosening exercise is to relax the mouth and leave it in a comfortable smiling position. Slowly drop the lower jaw as if a weight has been placed on the lower back teeth. Slowly lift the jaw and repeat the movement several times. Make sure the jaw only moves up and down, not from side to side.

The second exercise is described as ‘throwing the tongue away’, and it releases the tongue root using an action where the tongue flops out of the mouth with no muscular effort. The movement is to let the jaw ‘hang’ with the mouth in a relaxed smile, and flick the tongue out and back and then wiggle it from side to side without moving the jaw. You can use a mirror to check that you are doing this exercise correctly.

My jaw injury release involved my lower back molars suddenly dropping down what felt like two inches at the back of my mouth while I was performing these exercises, as my right jaw hinge dropped back down into its correct position. This staggeringly unexpected movement left me barely able to make goldfish mouth movements in shock as I tried to assimilate what had just happened in my mouth. It turned out to be the first step on the journey of a lifetime.

 The tongue root release action of throwing away the tongue mimics the flicker of a snake’s tongue as it warns of an impending attack. There are videos of Māori hakke ritual performances on YouTube which demonstrate the tongue action for this exercise.

Footnote references:

(1) The definitions of the brain’s constituent parts come from Neuroscience For Dummies   – Frank Amthor, PhD – (3rd Edition) – John Wiley and Sons, Inc – ISBN 978-1-394-17121-7.

(2) Science News Today – How The Brain Stores And Retrieves Memories (Editors’ article) (7th August 2025).

(3) Movement Professional – The Physiology of Fear – How Your Brain Responds To Threat (CrossFit MFP Collaboration) (8th September 2025).

(4) The excerpts from the article ‘Release Your Voice’ © 2000  by Barbara Alden of Vocal Constructivists are reproduced by kind permission of the author.

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